Role of Acetylation Processes in Pathogenesis with Complicated Gastric and Duodenal Ulcer
Sukhrob Alimov1, Kh. Karimov2, O. Teshayev3, O. Mavlyanov4, S. Alimov5, DJ. Mavlanov6
1SukhrobAlimov, Assistant Teacher, Ph.D. Researcher of the Surgeon Department of Tashkent Medical Academy, Tashkent, Uzbekistan,
2Kh. Karimov, Research Institute of Hematology and blood transfusion, Tashkent, Uzbekistan
3O. Teshayev, Surgeon department, Tashkent Medical Academy, Tashkent, Uzbekistan
4O. Mavlyanov, Surgeon department, Tashkent Medical Academy, Tashkent, Uzbekistan
5S. Alimov, Surgeon department, Tashkent Medical Academy, Tashkent, Uzbekistan
6DJ. Mavlanov, Surgeon department, Tashkent Medical Academy, Tashkent, Uzbekistan
Manuscript received on 26 October 2020 | Revised Manuscript received on 17 November 2020 | Manuscript Accepted on 15 December 2020 | Manuscript published on 30 December 2020. | PP: 1-7 | Volume-1 Issue-1, December 2020 | Retrieval Number: A1007061120 /2020©LSP
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© The Authors. Published by Lattice Science Publication (LSP). This is an open access article under the CC-BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/)
Abstract: Peptic ulcer is a chronic recurring polyetiological disease that occurs as a result of the interaction of exogenous and endogenous factors: a hereditary predisposition from 5.5 to 50%, the type of nervous system, endocrine systems, psychoemotional features, metabolic characteristics, biochemical reactions, immune status, cytokine profile and environmental factors, resulting in a violation between the factors of “aggression” and “protection” of the mucous membrane of the gastroduodenal zone. In the etiology of peptic ulcer, a certain role is played by hereditary burden. Therefore, when studying the nature of the hereditary predisposition to peptic ulcer of the stomach and duodenum, an important area is the identification of genetic markers.
Keywords: HP- Helecobaktery Pylori N-acetyltransferase GU- Gastric ulcer DU- Duodenal ulcer.
Scope of the Article: Duodenal ulcer