Role of Acetylation Processes in Pathogenesis with Complicated Gastric and Duodenal Ulcer
Sukhrob Alimov1, Kh. Karimov2, O. Teshayev3, O. Mavlyanov4, S. Alimov5, DJ. Mavlanov6

1SukhrobAlimov, Assistant Teacher, Ph.D. Researcher of the Surgeon Department of Tashkent Medical Academy, Tashkent, Uzbekistan.

2Kh. Karimov, Research Institute of Hematology and blood Transfusion, Tashkent, Uzbekistan.

3O. Teshayev, Surgeon Department, Tashkent Medical Academy, Tashkent, Uzbekistan.

4O. Mavlyanov, Surgeon Department, Tashkent Medical Academy, Tashkent, Uzbekistan.

5S. Alimov, Surgeon Department, Tashkent Medical Academy, Tashkent, Uzbekistan.

6DJ. Mavlanov, Surgeon Department, Tashkent Medical Academy, Tashkent, Uzbekistan.

Manuscript received on 26 October 2020 | Revised Manuscript received on 17 November 2020 | Manuscript Accepted on 15 December 2020 | Manuscript published on 30 December 2020. | PP: 1-7 | Volume-1 Issue-1, December 2020 | Retrieval Number: A1007061120 /2020©LSP

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Abstract: Peptic ulcer is a chronic recurring polyetiological disease that occurs as a result of the interaction of exogenous and endogenous factors: a hereditary predisposition from 5.5 to 50%, the type of nervous system, endocrine systems, psychoemotional features, metabolic characteristics, biochemical reactions, immune status, cytokine profile and environmental factors, resulting in a violation between the factors of “aggression” and “protection” of the mucous membrane of the gastroduodenal zone. In the etiology of peptic ulcer, a certain role is played by hereditary burden. Therefore, when studying the nature of the hereditary predisposition to peptic ulcer of the stomach and duodenum, an important area is the identification of genetic markers.

Keywords: HP- Helecobaktery Pylori N-acetyltransferase GU- Gastric ulcer DU- Duodenal ulcer.
Scope of the Article: Duodenal ulcer